章 急性胰腺炎與全身性炎癥反應(yīng)綜合征 1.Deoxycholic acid inhibited proliferation and induced apoptosis and necrosis byregulating the activity of transcription factors in rat pancreatic acinar cell lineAR42J(脫氧膽酸通過調(diào)節(jié)轉(zhuǎn)錄因子的活性抑制大鼠胰腺腺泡細(xì)胞系A(chǔ)R42J的增殖并誘導(dǎo)其凋亡和壞死) 2.Regulatory effects of apoptosis-associated speck-like protein on cytokines in theP388D1 macrophage-like cell line(凋亡相關(guān)斑點(diǎn)樣蛋白對 P388D1巨噬細(xì)胞樣細(xì)胞系細(xì)胞因子的調(diào)控作用) 3.CEBPE Promotes LPS-Induced IL-1B Transcription and Secretion in AlveolarMacrophages via NOD2 Signaling (CEBPB促進(jìn)LPS誘導(dǎo)的肺泡巨噬細(xì)胞IL-1p的轉(zhuǎn)錄和分泌) 4.Identification of Key Biomarkers Associated with Immunogenic Cell Death andTheir Regulatory Mechanisms in Severe Acute Pancreatitis Based on WGCNA andMachine Learning(基于WGCNA 和機(jī)器學(xué)習(xí)方法對重癥急性胰腺炎時免疫源性細(xì)胞死亡及 其調(diào)節(jié)機(jī)制相關(guān)的關(guān)鍵生物標(biāo)志物的確定) 5.Exosome-derived CIRP: An amplifier of inflammatory diseases(外泌體特異性CIRP;炎癥反應(yīng)的放大器) 6.Protective Effects of Emodin-Induced Neutrophil Apoptosis via the Ca*-Caspase12 Pathway against SIRS in Rats with Severe Acute Pancreatitis (大黃素誘導(dǎo)的中性粒細(xì)胞凋亡通過Ca*-Caspase 12途徑對重癥急性胰腺炎大鼠的SIRS 具有保護(hù)作用) 7.Inhibition of Necroptosis in Acute Pancreatitis: Screening for RIPK1 Inhibitors(急胰腺炎壞死性凋亡的抑制:RIPK1抑制劑的篩選) 8.Regulatory effects of emodin on NF-kB activation and infammatory cytokine expressiorin RAW 264.7 macrophages (大黃素對 RAW 264.7 巨噬細(xì)胞中NF-xB活化和炎癥因子表達(dá)的調(diào)節(jié)作用) 9.Qingyi Deces acute biliary pancreatitis by targeting Gpbar1NF-rb pathway (清胰湯靶向Gpbar1NF-rb通路緩解急性膽源性胰腺炎) 10.Effects of Qingyi decoction onammatory markers in patients with acute pancreatitis: Aeta-analysis (清胰湯對急性胰腺炎患者炎 性標(biāo)志物的影響:蕓萃分析) 第二章 急性胰腺炎腸道屏障損傷 1.Intestinal barrier damage, systemic inflammatory response syndrome, and acute lung in-jury: A troublesome trio for acute pancreatitis(腸道屏障損傷、全身性炎癥反應(yīng)綜合征和肺損傷:急性胰腺炎的三重奏) 2.The gut-lung axis in severe acute Pancreatitis-associated lung injury: The protection bythe gut microbiota through short-chain fatty acids(腸道菌群通過短鏈脂肪酸對急性胰腺炎肺損傷時腸-肺軸的保護(hù)作用) 3.Intestinal Microbiota - An Unmissable Bridge to Severe Acute Pancreatitis-AssociatedAcute Lung Injury (腸道菌群對急性胰腺炎 相關(guān)性肺損傷具有不可或缺的作用) 4.Therapeutic effect of Qingyi decoction in severe acute pancreatitis-induced intestinal bar-rier injury(清胰湯對重癥急性胰腺炎所致腸道屏障損傷的治療作用) 5.Qingyi decoction attenuates intestinal epithelial cell injury via the calcineurinnuclearfactor of activated T-cells pathway(清胰湯 通過NFAT 細(xì)胞通路減緩腸上皮細(xì)胞損傷) 第三章 急性胰腺炎肺損傷 1.Effects and mechanisms of alveolar type Il epithelial cell apoptosis in severe pancreatitis-induced acute lung injury (肺泡I型上皮細(xì) 胞凋亡在重癥急性胰腺炎所致急性肺損傷中的作用和機(jī)制) 2.Enhancement of ICAM-I via the JAK2STAT3 signaling pathway in a rat model of severeacute pancreatitis-associated lung injury (JAK2STAT3 信號通路在重癥急性胰腺炎相關(guān)肺損傷大鼠模型中對ICAM-I的增強(qiáng)效應(yīng)) 3.Inhibition of SOCs Attenuates Acute Lung Injury Induced by Severe Acute Pancreatitis inRats and PMVECs Injury Induced by Lipopolysaccharide(抑制鈣庫操縱性鈣離子通道可減輕大鼠重癥急性胰腺炎肺損傷和脂多糖所致肺血管內(nèi)皮細(xì)胞損傷) Effect of angiopoietin-like protein 4 on rat pulmonary microvascular endothelial cells ex-posed to LPS(血管生成素樣蛋白4對 LPS 所致大鼠肺微血管內(nèi)皮細(xì)胞的影響) 4.Effects of hepoxia-inlucible factor-lor andatrix metalloproteinase-9 on alveolar-capillary barrier disru ung edemaodels of severe acute pancreatitis associated lung injury (缺氧誘導(dǎo)因子-1c和基質(zhì)金屬蛋白酶-9對重癥急性胰腺炎相關(guān)肺損傷大鼠肺泡-毛細(xì)血管屏障破壞和 肺水腫的影響) 6.Multifunctional neuron-specific enolase: its ro
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